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1. It is estimated that a newly diagnosis type 2 diabetes patient has actually had the disease for:

A. 6 months B. 2 years C. 5 years D. 9 years

2. How much pancreatic b-cell function is already lost at the time of diagnosis for type 2 diabetes?

A. < 10% B. 20-40% C. 40-60% D. >80%

3. Which of the following is not a dysfunctional defect that leads to hyperglycemia and type 2 diabetes?

A. Increased glucose reabsorption in the kidneys B. Increase glucose uptake to the peripheral tissue C. Increased hepatic glucose production D. Increased pancreatic alpha-cell secretion

4. Which dysfunctional defects does insulin “fix”?

A. Fat, liver, muscle, pancreatic alpha cell and beta cell B. Fat, kidney, liver, muscle and pancreatic beta cell C. Brain, GI tract, liver, pancreatic alpha cell and beta cell D. GI tract, liver, muscle, pancreatic alpha cell and beta cell

5. Which of the following is a basal insulin?

A. Aspart U-100 B. Degludec U-200 C. Lispro U-200 D. Regular U-500

6. Which of the following would NOT be an appropriate “add-on” therapy to optimized basal insulin?

A. Exenatide twice daily B. Liraglutide once daily C. Lispro U100 once daily D. Regular U-500 twice daily

Maintenance is scheduled for 8/7/2023 7:00 AM EST - 8:00 AM EST.

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1. It is estimated that a newly diagnosis type 2 diabetes patient has actually had the disease for:

2. How much pancreatic b-cell function is already lost at the time of diagnosis for type 2 diabetes?

3. Which of the following is not a dysfunctional defect that leads to hyperglycemia and type 2 diabetes?

4. Which dysfunctional defects does insulin “fix”?

5. Which of the following is a basal insulin?

6. Which of the following would NOT be an appropriate “add-on” therapy to optimized basal insulin?

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Maintenance is scheduled for 8/7/2023 7:00 AM EST - 8:00 AM EST.

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1. It is estimated that a newly diagnosis type 2 diabetes patient has actually had the disease for:

2. How much pancreatic b-cell function is already lost at the time of diagnosis for type 2 diabetes?

3. Which of the following is not a dysfunctional defect that leads to hyperglycemia and type 2 diabetes?

4. Which dysfunctional defects does insulin “fix”?

5. Which of the following is a basal insulin?

6. Which of the following would NOT be an appropriate “add-on” therapy to optimized basal insulin?

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